Nanette Nascone-Yoder

Bio

Nanette Nascone-Yoder obtained her BS in Molecular Biology from Eckerd College, and Ph.D. in Cell and Developmental Biology at Harvard University.

Dr. Nascone-Yoder established her laboratory at NC State’s College of Veterinary Medicine in 2006 and was selected as a University Faculty Scholar in 2013. As an Associate Professor in the Department of Molecular Biomedical Sciences, Dr. Nascone-Yoder’s research is at the intersection of development, evolution and toxicology. Her lab focuses on discovering the cellular and molecular mechanisms by which organs become left-right asymmetric, as a way to understand the causes of laterality-related birth defects. The Nascone-Yoder lab recently pioneered the use of a novel model organism for the study of organogenesis, the Budgett’s frog. The lab’s research has been funded by the National Science Foundation, National Institutes of Health and American Heart Association, and recommended by Faculty of 1000.

Publications

• Developmental regulation of cellular metabolism is required for intestinal elongation and rotation , DEVELOPMENT (2024)
• Morphoelastic models discriminate between different mechanisms of left-right asymmetric stomach morphogenesis , CELLS & DEVELOPMENT (2024)
• The people behind the papers - Julia Grzymkowski and Nanette Nascone-Yoder , DEVELOPMENT (2024)
• Rare variants in CAPN2 increase risk for isolated hypoplastic left heart syndrome , HUMAN GENETICS AND GENOMICS ADVANCES (2023)
• Normal Table of Xenopus development: a new graphical resource , DEVELOPMENT (2022)
• Single-minded 2 is required for left-right asymmetric stomach morphogenesis , DEVELOPMENT (2021)
• The twists and turns of left-right asymmetric gut morphogenesis , DEVELOPMENT (2020)
• Exome sequencing of family trios from the National Birth Defects Prevention Study: Tapping into a rich resource of genetic and environmental data , BIRTH DEFECTS RESEARCH (2019)
• Vangl2 coordinates cell rearrangements during gut elongation , DEVELOPMENTAL DYNAMICS (2019)
• The left-right asymmetry of liver lobation is generated by Pitx2c-mediated asymmetries in the hepatic diverticulum , DEVELOPMENTAL BIOLOGY (2018)